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nikon biostation ct  (Nikon)


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    Nikon nikon biostation ct
    Nikon Biostation Ct, supplied by Nikon, used in various techniques. Bioz Stars score: 95/100, based on 272 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/biostation+ct+system/BioStation+CT/pmc12854672-133-21-21
    Average 95 stars, based on 272 article reviews
    nikon biostation ct - by Bioz Stars, 2026-09
    95/100 stars

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    Related Articles

    other:

    Article Title: Enhancement of Cellular Adhesion and Proliferation in Human Mesenchymal Stromal Cells by the Direct Addition of Recombinant Collagen I Peptide to the Culture Medium
    Article Snippet: As already described, we analyzed cell adhesion to the surface of the wells using a BioStation CT system (Nikon).

    Article Title: Enhancement of Cellular Adhesion and Proliferation in Human Mesenchymal Stromal Cells by the Direct Addition of Recombinant Collagen I Peptide to the Culture Medium
    Article Snippet: In this study, we evaluated the effect of RCP on cell adhesion under different culture conditions, with RCP and without RCP in the medium using time lapse images recorded in a BioStation CT system (Nikon, Tokyo, Japan).

    Article Title: Pharmacologic Targeting of Histone H3K27 Acetylation/BRD4-dependent Induction of ALDH1A3 for Early-phase Drug Tolerance of Gastric Cancer
    Article Snippet: Cell 227 behavior was then monitored with a BioStation CT system (Nikon, Tokyo, Japan) under the 228 following conditions: picture position of tiling, 3 × 3; ×10 magnification, ch2 (Ex/Em 229 438/483), 800 ms of exposure time and 240 luminance; scheduling 0, 12, 36, and 60 hours.

    Article Title: Lung injury induces alveolar type 2 cell hypertrophy and polyploidy with implications for repair and regeneration
    Article Snippet: 48 hours post-isolation, media was changed and adherent cells were imaged every 15-minutes on a Biostation-CT system (Nikon Instruments, New York, USA) for a total of 5 hours.

    Article Title: Pharmacologic Targeting of Histone H3K27 Acetylation/BRD4-dependent Induction of ALDH1A3 for Early-phase Drug Tolerance of Gastric Cancer
    Article Snippet: Cell behavior was then monitored with a BioStation CT system (Nikon) under the following conditions: picture position of tiling, 3 × 3; × 10 magnification, ch2 (Ex/Em 438/483), 800 ms of exposure time and 240 luminance; scheduling 0, 12, 36, and 60 hours.

    Imaging:

    Article Title: Neuroblastoma-associated chromosomal aberrations drive cell identity loss in human neural crest via disruption of developmental regulators
    Article Snippet: .. Imaging started 24 hours after plating using the BioStation CT system (Nikon). ..

    Article Title: HDL nanoparticles have wound healing and anti-inflammatory properties and can topically deliver miRNAs
    Article Snippet: High content imaging was performed on the Nikon Biostation CT system purchased with the support of NIH 1S10OD021704-01. .. High content imaging was performed on the Nikon Biostation CT system purchased with the support of NIH 1S10OD021704-01. ..

    Isolation:

    Article Title: Lung Injury Induces Alveolar Type 2 Cell Hypertrophy and Polyploidy with Implications for Repair and Regeneration
    Article Snippet: .. Forty-eight hours after isolation, medium was changed, and adherent cells were imaged every 15 minutes on a Biostation-CT system (Nikon Instruments) for a total of 5 hours. .. Image sequences were analyzed in Fiji (ImageJ version 2.1.0/1.53c).



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    Generation and functional characterization of highly invasive CT-26 (HI CT-26) cells. ( A ) Schematic workflow for generating HI CT-26 cells. CT-26 cells stably expressing EGFP were injected into mice via the tail vein. After 7 days, liver tumor tissues were isolated, sorted by flow cytometry, and reinjected into recipient mice. This cycle was repeated three times to obtain HI CT-26 cells. ( B , D ) Transwell assay demonstrating enhanced migratory capacity of HI CT-26 cells compared to controls, imaged via bright-field/fluorescence microscopy ( B ), with corresponding quantitative analysis ( D ). ( C , E ) Invasion assay showing significantly enhanced invasive potential of HI CT-26 cells compared to controls, with representative bright-field images ( C ) and corresponding quantitative analysis ( E ). Quantitative analysis of the Transwell migration and invasion assays, respectively. **Data are presented as mean ± SD; n = 3 independent experiments. Statistical significance was determined by unpaired two-tailed Student’s t-test (* p < 0.05, ** p < 0.01). ( F <t>)</t> <t>Live-cell</t> tracking over 24 h revealed prolonged movement trajectories and higher motility in HI CT-26 cells. ( G ) Transendothelial migration assay: HI CT-26 cells crossed the HUVEC endothelial barrier within ~ 6 h, whereas < 60% of control cells completed transmigration within 12 h (see Movies 1–2, which vividly illustrate the accelerated migration speed and distinct morphological dynamics of HI CT-26 cells compared to controls. (H) Western blot analysis of EMT markers: HI CT-26 cells exhibited reduced E-cadherin and elevated N-cadherin, Vimentin, and Snail levels
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    Image Search Results


    Generation and functional characterization of highly invasive CT-26 (HI CT-26) cells. ( A ) Schematic workflow for generating HI CT-26 cells. CT-26 cells stably expressing EGFP were injected into mice via the tail vein. After 7 days, liver tumor tissues were isolated, sorted by flow cytometry, and reinjected into recipient mice. This cycle was repeated three times to obtain HI CT-26 cells. ( B , D ) Transwell assay demonstrating enhanced migratory capacity of HI CT-26 cells compared to controls, imaged via bright-field/fluorescence microscopy ( B ), with corresponding quantitative analysis ( D ). ( C , E ) Invasion assay showing significantly enhanced invasive potential of HI CT-26 cells compared to controls, with representative bright-field images ( C ) and corresponding quantitative analysis ( E ). Quantitative analysis of the Transwell migration and invasion assays, respectively. **Data are presented as mean ± SD; n = 3 independent experiments. Statistical significance was determined by unpaired two-tailed Student’s t-test (* p < 0.05, ** p < 0.01). ( F ) Live-cell tracking over 24 h revealed prolonged movement trajectories and higher motility in HI CT-26 cells. ( G ) Transendothelial migration assay: HI CT-26 cells crossed the HUVEC endothelial barrier within ~ 6 h, whereas < 60% of control cells completed transmigration within 12 h (see Movies 1–2, which vividly illustrate the accelerated migration speed and distinct morphological dynamics of HI CT-26 cells compared to controls. (H) Western blot analysis of EMT markers: HI CT-26 cells exhibited reduced E-cadherin and elevated N-cadherin, Vimentin, and Snail levels

    Journal: Cancer Cell International

    Article Title: The notch-miR-188-5p-TIMP2/3 axis orchestrates exosome-driven pre-metastatic niche formation in colorectal cancer

    doi: 10.1186/s12935-025-04164-4

    Figure Lengend Snippet: Generation and functional characterization of highly invasive CT-26 (HI CT-26) cells. ( A ) Schematic workflow for generating HI CT-26 cells. CT-26 cells stably expressing EGFP were injected into mice via the tail vein. After 7 days, liver tumor tissues were isolated, sorted by flow cytometry, and reinjected into recipient mice. This cycle was repeated three times to obtain HI CT-26 cells. ( B , D ) Transwell assay demonstrating enhanced migratory capacity of HI CT-26 cells compared to controls, imaged via bright-field/fluorescence microscopy ( B ), with corresponding quantitative analysis ( D ). ( C , E ) Invasion assay showing significantly enhanced invasive potential of HI CT-26 cells compared to controls, with representative bright-field images ( C ) and corresponding quantitative analysis ( E ). Quantitative analysis of the Transwell migration and invasion assays, respectively. **Data are presented as mean ± SD; n = 3 independent experiments. Statistical significance was determined by unpaired two-tailed Student’s t-test (* p < 0.05, ** p < 0.01). ( F ) Live-cell tracking over 24 h revealed prolonged movement trajectories and higher motility in HI CT-26 cells. ( G ) Transendothelial migration assay: HI CT-26 cells crossed the HUVEC endothelial barrier within ~ 6 h, whereas < 60% of control cells completed transmigration within 12 h (see Movies 1–2, which vividly illustrate the accelerated migration speed and distinct morphological dynamics of HI CT-26 cells compared to controls. (H) Western blot analysis of EMT markers: HI CT-26 cells exhibited reduced E-cadherin and elevated N-cadherin, Vimentin, and Snail levels

    Article Snippet: The migration process was dynamically monitored using a live-cell imaging workstation (Nikon BioStation CT) with phase-contrast microscopy and multi-channel fluorescence imaging.

    Techniques: Functional Assay, Stable Transfection, Expressing, Injection, Isolation, Flow Cytometry, Transwell Assay, Fluorescence, Microscopy, Invasion Assay, Migration, Two Tailed Test, Cell Tracking Assay, Control, Transmigration Assay, Western Blot